Download PDF by Fima Lifshitz: Clinical Pediatric Endocrinology, Fifth Edition

By Fima Lifshitz

ISBN-10: 0470987111

ISBN-13: 9780470987117

ISBN-10: 1405126558

ISBN-13: 9781405126557

This new version of the benchmark textual content on scientific pediatric endocrinology nonetheless is still on the vanguard of global scientific opinion. in addition, the fashion, which has been the hallmark of the e-book for the final two decades, has been maintained for this 5th Edition.

New beneficial properties of this 5th variation include:


  • Special new chapters disguise the fetal origins of grownup affliction and the
    endocrine outcomes and administration of serious illness
  • Now opens with a uncomplicated technological know-how part summarizing the mechanisms of hormone motion, genomics, proteomics and the appliance of molecular biology to medical practice
  • Two new co-editors were brought - Peter Clayton and Rosalind Brown
  • Every bankruptcy has been updated

Content:
Chapter 1 ideas of Hormone motion (pages 1–17): Melissa Westwood
Chapter 2 Genetics, Genomics, Proteomics, and Bioinformatics (pages 18–44): Peter Kopp
Chapter three Measuring Hormones, Molecular checks, and Their medical software (pages 45–66): Jan M. Wit and Marcel Karperien
Chapter four Congenital issues of the Hypothalamic?Pituitary Axis (pages 67–89): Ameeta Mehta and Mehul T. Dattani
Chapter five general and Disordered progress (pages 90–112): Leena Patel and Peter E. Clayton
Chapter 6 Genetic Syndromes and Dysmorphology (pages 113–123): Jennifer Batch
Chapter 7 got Abnormalities of the Hypothalamic?Pituitary Axis resulting in progress Impairment (pages 124–152): Dennis J. Brenner and Edward O. Reiter
Chapter eight improvement of the Reproductive platforms (pages 153–170): John C. Achermann
Chapter nine Ambiguous Genitalia (pages 171–182): Ieuan A. Hughes
Chapter 10 common and irregular Puberty (pages 183–210): Mehul T. Dattani and Peter C. Hindmarsh
Chapter eleven Gynecology (pages 211–217): Sarah M. Creighton
Chapter 12 The Thyroid and its issues (pages 218–253): Rosalind S. Brown and Stephen Huang
Chapter thirteen The Parathyroid and issues of Calcium Metabolism (pages 254–279): Jeremy Allgrove
Chapter 14 issues of Bone Metabolism (pages 280–292): Ingrid A. Holm
Chapter 15 The Adrenal Cortex and its issues (pages 293–351): Walter L. Miller
Chapter sixteen Polyglandular Syndromes (pages 352–366): Catherine J. Owen, Tim D. Cheetham and Simon H.S. Pearce
Chapter 17 problems of Water stability (pages 367–395): Rebecca P. eco-friendly, Joseph A. Majzoub and Louis J. Muglia
Chapter 18 Endocrine Programming and the Fetal and Early?Life Origins of grownup illness (pages 396–409): Caroline H.D. Fall
Chapter 19 Weight legislation and Monogenic weight problems (pages 410–418): I. Sadaf Farooqi
Chapter 20 Metabolic outcomes of weight problems and Their administration (pages 419–435): Michael Freemark
Chapter 21 Diabetes Mellitus (pages 436–473): Andrew W. Norris and Joseph I. Wolfsdorf
Chapter 22 Hypoglycemia (pages 474–491): Khalid Hussain and Mark J. Dunne
Chapter 23 Endocrine results of Systemic affliction: severe affliction (pages 492–504): Greet Van Den Berghe
Chapter 24 outcomes of Radiotherapy, Chemotherapy, and Bone Marrow Transplant (pages 505–522): Helena A. Davies
Chapter 25 checks and common Values in Pediatric Endocrinology (pages 523–564): Lesley J. Tetlow and Peter E. Clayton

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Example text

Genes can be defined as functional units that are regulated by transcription and encode RNA (Fig. 2). The majority of RNA transcripts consists of mRNA, which is subsequently translated into protein. Other RNA transcripts exert specialized functions, such as transfer of amino acids for polypeptide synthesis (tRNA), contribute to ribosome structure (rRNA), or regulate transcription. Genes account for 10–15% of the genomic DNA. Much of the remaining DNA consists of highly repetitive sequences, the function of which remains poorly understood.

For example, monoallelic mutations in the transcription factor HESX1 (RPX) result in various constellations of pituitary hormone deficiencies, and the phenotype is variable among family members with the same mutation. 2). 26 RNA Pol II Fig. 3. Control of gene transcription by the ligand-dependent thyroid hormone receptor (TR) interacting with co-repressors and co-activators. In the absence of triiodothyronine (T3), the TR binds to a thyroid hormone response element (TRE) in conjunction with the retinoid X receptor (RXR).

Mullis, Nobel prize 1998). PCR provides a rapid way of selectively amplifying specific DNA fragments in vitro (Fig. 4). In a first step, DNA is denatured by heat. This is followed by annealing of oligonucleotides (primers). Primers are complementary to the opposite strands and flank the DNA region to be amplified. A heat-resistant polymerase then extends the primers. Repeated cycles of this process lead to an exponential amplification of the region of interest. This amplicon can then be used for further analyses, for example sequence analysis.

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Clinical Pediatric Endocrinology, Fifth Edition by Fima Lifshitz


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